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These results imply that a properly functioning endothelium controls the potential harmful effects of Hcy through the release of EDRF and the formation of S-NO-Hcy

Pathogenesis of asthma Airway inflammation in asthma is a complex process involving the interactions between immunological mediators produced by inflammatory cells such as mast cells, eosinophils, basophils, neutrophils, dendritic cells and lymphocytes [24]

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It is involved in the formation of neurotransmitters, heart and endothelial dysfunction, the immune response, and pain sensitivity [1]